No significant differences between deaf and hearing individuals were found in target visual and auditory areas when the motion and form components of the stimuli were isolated (contrasted with a static visual image). However, increases in activation were found in the deaf group in the superior temporal gyrus (BA 22 and 42) and in an area located at the junction of the parieto-occipital sulcus and the calcarine fissure (encompassing parts of the cuneus, precuneus and the lingual gyrus) for the GMS and FFM conditions as
well as for the static image, relative to a baseline condition absent of any visual stimulation. These results suggest that the observed cross-modal recruitment of auditory areas in deaf individuals does not appear to be specialized for motion processing, but rather is present learn more for both motion and static visual stimuli. (C) 2013 IBRO. Published by Elsevier Ltd. All rights reserved.”
“Data showing how schizophrenia patients tend to be more susceptible to false memories have been rather mixed and, AZD7762 solubility dmso as far as we know, no studies have investigated whether patients with a schizophrenia spectrum disorder are particularly prone to imagination inflation effects, that is, whether
repeatedly imagining an action increases the likelihood of remembering the action as having been performed. In this study, a group of patients with psychosis and a group of normal controls were asked to perform or to imagine performing simple action statements one or four Sclareol times in a single study session. In a test session that occurred 24
h later, participants were instructed to discriminate whether the action statement had been carried out, imagined or whether it was new (a source monitoring task).
The primary finding was that patients were more susceptible to source-monitoring errors than controls, especially in terms of considering an imagined action as having been performed. However, both groups showed comparable levels of imagination inflation effects. Results add evidence to the hypothesis that the nature of patients’ false memories may be particularly linked to poor use of source-monitoring processes. (C) 2009 Elsevier Ireland Ltd. All rights reserved.”
“The present study examined whether nicotinic acetylcholine receptors (nAChRs) of the CA1 regions of the dorsal hippocampus and medial septum (MS) are involved in cross state-dependent memory retrieval between WIN55, 212-2 (WIN, a non-selective CB1/CB2 receptor agonist) and nicotine or ethanol. Memory retrieval was measured in one-trial step-down type passive avoidance apparatus in male adult mice. Pre-training intraperitoneal administration of WIN (0.1-1 mg/kg) dose-dependently impaired memory retrieval when it was tested 24 h later.