Monthly Archives: March 2018

Even if one considers the large size of the B. japonicum genome (9.1 Mbp), the number of 24 putative RND-type transporters it encodes is notably high. Yet, none of the defects in the ΔbdeAB strain could apparently be rescued by its … Continue reading →

CSP gene analysis based on the repeat patterns showed similar results that the sequences from the imported cases well matched with the patient’s traveled countries and completely discriminated with indigenous cases. AMA-1 gene analysis also supported these results. We were … Continue reading →

subtilis and Escherichia coli; however, the precise manner of SpoIISA toxicity remains unknown. In this work, we focused on the N-terminal, transmembrane domain of SpoIISA and verified the prediction of its topology. Using truncated SpoIISA constructs, we show that the … Continue reading →

1). A region containing four prolines was found between positions 176 and 182; only one other sequence – that of gp48 from the Mycobacterium phage Puhltonio – also had such a proline-rich region (Fig. 1). Because proline residues are conformationally … Continue reading →

, 2000, 2001). The N-terminal domain of Bcy1 served to target it properly during logarithmic and stationary phase (Griffioen et al., 2000). Phosphorylation of its N-terminal domain directed Bcy1 to cytoplasm. Bcy1 modification was found to be dependent on Yak1 kinase … Continue reading →

, 2000, 2001). The N-terminal domain of Bcy1 served to target it properly during logarithmic and stationary phase (Griffioen et al., 2000). Phosphorylation of its N-terminal domain directed Bcy1 to cytoplasm. Bcy1 modification was found to be dependent on Yak1 kinase … Continue reading →