(C) 2013 Phytochemical Society of Europe. Published by Elsevier B.V. All rights reserved.”
“BACKGROUND: Electrolytic manganese residues are the largest and most dangerous waste stream from the electrolytic manganese metal industry, and
recovery of Mn from manganese residues is one of the main methods of recycling this waste. Bioleaching of electrolytic manganese residues for Mn extraction was investigated in this IPI-145 cost study.
RESULTS: An extraction efficiency as high as 99.7% was achieved at 1% slag pulp concentration, 2320 mg L(-1) extraction concentration and 80% extraction efficiency was attained at 8% slag pulp concentration, indicating the application potential Selleck Panobinostat of bioleaching in Mn extraction from manganese residues.
CONCLUSION: The extraction of Mn from manganese residues depended exclusively on the non-contact mechanism. The acidic dissolution of soluble Mn(2+)
induced by sulfur-oxidizing bacteria resulted in 91.9% Mn extraction, while the reduction dissolution of insoluble Mn(4+) by iron-oxidizing bacteria caused 5.8% Mn extraction. The combined action of sulfur-oxidizing bacteria and iron-oxidizing bacteria assured the maximum Mn extraction. EDS and XRD analysis of bioleaching residues further demonstrated the bioleaching mechanism involved. (C) 2011 Society of Chemical Industry”
“Background-Recent trial data Panobinostat have challenged the hypothesis that cholesteryl ester transfer protein (CETP) and high-density lipoprotein cholesterol (HDL-C) have causal roles in atherothrombosis. One method to evaluate this issue is to examine whether polymorphisms in the CETP gene that impact on HDL-C levels also impact on the future development of myocardial infarction.
Methods and Results-In
a prospective cohort of 18 245 initially healthy American women, we examined over 350 000 singe-nucleotide polymorphisms (SNPs) first to identify loci associated with HDL-C and then to evaluate whether significant SNPs within these loci also impact on rates of incident myocardial infarction during an average 10-year follow-up period. Nine loci on 9 chromosomes had 1 or more SNPs associated with HDL-C at genome-wide statistical significance (P < 5 x 10(-8)). However, only SNPs near or in the CETP gene at 16q13 were associated with both HDL-C and risk of incident myocardial infarction (198 events). For example, SNP rs708272 in the CETP gene was associated with a per-allele increase in HDL-C levels of 3.1 mg/dL and a concordant 24% lower risk of future myocardial infarction (age-adjusted hazard ratio, 0.76; 95% CI, 0.62 to 0.94), consistent with recent meta-analysis. Independent and again concordant effects on HDL-C and incident myocardial infarction were also observed at the CETP locus for rs4329913 and rs7202364. Adjustment for HDL-C attenuated but did not eliminate these effects.